Vaughan & Asbury's General Ophthalmology, 18th Edition (LANGE Clinical Medicine)
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The most concise, current, and comprehensive overview of general ophthalmology – extensively revised and updated
A Doody's Core Title for 2015!
For five decades, General Ophthalmology has offered authoritative, state-of-the-art coverage of the diagnosis and treatment of all major ophthalmic diseases, as well as neurological and systemic diseases causing visual disturbances. The eighteenth edition builds on this tradition of excellence by offering an increased number of color illustrations, new chapters, and major revisions of existing chapters – to deliver all-inclusive coverage that encompasses pathophysiology, basic science, and the latest clinical perspectives.
A must-read for medical students, ophthalmology residents, practicing ophthalmologists, nurses, optometrists, clinicians in other fields of medicine and surgery, and health-related professionals, the eighteenth edition of General Ophthalmology features:
- New chapters on Ophthalmic Emergencies, Causes and Prevention of Vision Loss, Vision Rehabilitation, and Functional Vision Score
- A glossary of terms relating to the eye
- Contributions from more than thirty international experts
- Extensive updates and revisions throughout to reflect the latest findings and advances
years of age, and more than 50% have associated cardiovascular disease. Predisposing factors and investigations are discussed in Chapter 15. Chronic open-angle glaucoma should always be excluded (see Chapter 11). The major complications associated with retinal vein occlusion are reduced vision from macular edema, neovascular glaucoma secondary to iris neovascularization, and retinal neovascularization. 1. MACULAR EDEMA IN RETINAL VEIN OCCLUSION Macular dysfunction occurs in almost all
patients with advanced glaucoma. The main side effects are a rise in pressure for 1–4 hours in about one-third of eyes, usually preventable by pretreatment with apraclonidine drops, and a rise in pressure for 1–3 weeks in about 2% of treated eyes. Initial treatment with 50 laser burns in 180° of the trabecular meshwork, followed by treatment to the other 180° at a later date if necessary, reduces the severity of these pressure rises. Subsequent loss of pressure control can be very sudden after
visual performance, that is, visual acuity not correctable with conventional glasses or contact lenses. They may have cloudy vision, constricted fields, or large scotomas. There may be additional functional complaints: glare sensitivity, abnormal color perception, or diminished contrast. Some patients have diplopia. A frequent complaint is confusion from overlapping but dissimilar images from each eye. The term “low vision” covers a wide range. A person in the early stages of an eye disease may
or more sessions, and possibly a loaner lens for home or job trial. Older patients usually need more adaptation time and reinforcement than younger or congenitally impaired persons. Practitioners and staff benefit from training programs to learn how to manage a low-vision patient in the office. Basic setups for incorporating low vision into a practice are reviewed in a number of publications. Instruction is the key to success in vision rehabilitation. Over 90% of patients succeed with
therapy is used occasionally to treat perineural invasion into bone or the orbit, and exenteration is generally performed in cases with extensive eyelid destruction or massive orbital invasion. All patients with squamous cell carcinoma of the eyelid should be advised of the risk of residual or recurrent tumor post-treatment and encouraged to adhere to recommended follow up. Figure 4–14. Squamous cell carcinoma, with a typical nodular shape and ulcerated center, of the upper eyelid. (Courtesy