Ridley's The Vulva
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Bridging the gap between dermatology and gynaecology in the study of vulval diseases, this new edition is an exceptional reference text, offering the most up-to-date guidance on diagnosis and management.
The last 10 years have seen an enormous increase in interest in genital skin disease along with a much needed expansion in the number of clinics dedicated to the diagnosis and treatment of vulval disorders. This new third edition of Marjorie Ridley’s The Vulva contains all the topics covered in the original book, but now includes the many advances that have been made since the last publication.
Now entitled Ridley’s The Vulva, this is a comprehensive textbook that specialises in the diagnosis and management of this wide-ranging area. Many chapters have been extensively revised, and illustrations are all now in full colour, significantly enhancing some of the detail of both the clinical and histological appearances.
was not recognized by others for a considerable time. In the USA, Taussig (1929) also contributed to the debate; he described a leukoplakic vulvitis, which began as a hypertrophic condition but ended as an atrophic one, with an inflammatory infiltrate and a subepidermal ‘collagenous’ zone. He used the term kraurosis for the end stage of this leukoplakic vulvitis. X-ray therapy was one of the favoured treatments of leukoplakic vulvitis in the early days, which may have been a factor in the
6th edn. Arnold, London. King, C.R., Magenis, E. & Bennett, S. (1978) Pregnancy and the Turner syndrome. Obstetrics and Gynecology 52, 617– 624. Koff, A.K. (1933) Development of the vagina in the human fetus. Contributions to Embryology 24, 59–90. Kohn, G., Yarkonis, S. & Cohen, M.M. (1980) Two conceptions in a 45X woman. American Journal of Medical Genetics 5, 339–343. Krantz, K.E. (1977) The anatomy and physiology of the vulva and vagina. In: Scientific Foundation of Obstetrics and Gynaecology,
histology. Some histological features such as pale, swollen keratinocytes, basal cell vacuolation and basal layer destruction with subepidermal cleavage are related to the use of this preparation and may lead to misdiagnosis (Cazes et al. 2007). Ultrastructural changes may also be attributable to EMLA and were mistaken for lysosomal storage disease in one series (Vallance et al. 2004). Specimens for immunofluorescence are, after washing in saline, put into liquid nitrogen or into transport
between tuberculosis and HIV infection is well known, and genitourinary tuberculosis is more common in HIV-positive patients. In primary infection, the initial lesion is an inconspicuous brown-red papule, but this is often missed so that the clinical picture is dominated by inguinal or femoral adenitis. The primary tuberculous lesion usually heals after a few months, but the enlarged glands may persist and break down. In other forms of vulval tuberculosis, nodules appear which develop into ulcers
chromosome (Blagowidow et al. 1989) and/or the X chromosome (Scherer et al. 1989). Clitoral enlargement is not uncommon, but the most important aspect of this condition is the increased incidence of gonadal neoplasms (Simpson 1982). Bilateral gonadectomy is therefore indicated as a prophylactic measure (Dewhurst 1980, Grumbach & Conte 1992). In all forms of ovarian dysgenesis oestrogen replacement therapy is recommended at 12–13 years of age, eventually to be cycled monthly with progesterone