Pathophysiology Flash Cards (Lange Flash Cards)

Pathophysiology Flash Cards (Lange Flash Cards)

Language: English

Pages: 544

ISBN: 0071767401

Format: PDF / Kindle (mobi) / ePub


Pathophysiology Flash Cards contain 240 clinical cases that challenge you to apply principles to real life scenarios.

  • Each two-sided card features a clinical case and challenges you to diagnose the condition
  • Provides a clear explanation of the underlying physiology
  • Allows you to brush up on system-specific disorders
  • A super-effective way to prepare for pathophysiology courses and board exams
  • Student and peer reviewed for the most relevant material possible

Minor Emergencies (3rd Edition)

Clinical Hepatology: Principles and Practice of Hepatobiliary Diseases: Volume 1

Junqueira's Basic Histology, Text and Atlas (13th Edition)

The Washington Manual of Medical Therapeutics (35th Edition) (Lippincott Manual Series)

How to Learn & Memorize Medical Terminology

Antimicrobials: Synthetic and Natural Compounds

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

intestinal fluid secretion, decreased fluid absorption, increased motility, and increased luminal osmolarity. Common infectious agents include, but are not limited to, viruses (e.g., noroviruses, rotaviruses), bacteria (e.g., Shigella, Salmonella, Campylobacter, Yersinia, Clostridia, Listeria, Vibrio, E. coli), protozoa (e.g., Giardia, Cryptosporidium, Entamoeba), and fungi. C. difficile infection is the most common causative agent in hospitalized patients. Prior antibiotic therapy disrupts the

Hypopituitarism This patient has anterior hypopituitarism. There is decreased secretion of more than one of the anterior pituitary tropins. The condition may be caused by lesions to the hypothalamus, pituitary stalk, or pituitary gland. Hypopituitarism can be caused by mass lesions including pituitary tumors, brain tumors, granulomas, or cysts, or may occur without mass lesions in cases such as trauma, ischemia, radiation treatment, or congenital syndromes. The presence of headache and visual

represent the aberrant atrial impulse exciting the ventricle prematurely, without normal AV nodal control. Sudden death in this condition is rare. The condition is often seen in young people, and ablation of the accessory pathway will definitively treat the condition. A 77-year-old female is evaluated in a physician’s office for symptoms of fatigue, nausea/vomiting, and visual disturbances. She states that she has been having these symptoms for the past 2 weeks and has felt progressively worse

shows absence of HbAj with increased HbA2 and elevated fetal hemoglobin (HbF). β-Thalassemia Major (β-Thalassemia Minor) This child has β-thalassemia major, confirmed by the absence of HbA1 (composed of a-2 and β-2) on electrophoresis. In contrast to heterozygous β-thalassemia minor, the patient is homozygous for deletion of the β-hemoglobin gene. Therefore, absence of β-chain synthesis increases formation of HbA2 (α-2, δ-2) and HbF (α-2, γ-2). The anemia is a result of ineffective

irregular Borders, heterogeneity of Color, large Diameter, and Enlarging size. The melanoma cells start proliferating superficially within the dermis and epidermis in the radial growth phase. However, they gain invasive potential and initiate the vertical growth phase. There are hereditary syndromes that predispose to melanomas, such as xeroderma pigmentosum (XP). XP occurs secondary to genetic mutation involved in post ultraviolet (UV) radiation DNA repair. It is important to initiate treatment

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